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Even if the usage of E2 as a therapeutic agent for human use is controversial, targeting specific ERs in the cardiovascular system could open new and perhaps safer therapeutic options for using E2 to protect the cardiovascular system [5] (Figure 1)

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These α-keto acids further get metabolised to either three or four carbon units which by gluconeogenesis help produce glucose as a source of energy or can be broken down directly to CO 2 and H 2 O and produce energy