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crucially, it does not bind or activate the GH receptor and does not induce cell proliferation, distinguishing it mechanistically from both hGH and GLP-1 class compounds ( In murine obesity models, chronic administration reduces adipose tissue mass and increases plasma glycerol (lipolysis index) through upregulation of β3-adrenergic receptor (β3-AR) mRNA expression in fat tissue, while β3-AR knockout mice are largely unresponsive to its chronic lipolytic effects ( The peptide also acutely increases fat oxidation and energy expenditure via β3-AR-independent pathways, suggesting multiple lipolytic mechanisms