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Although the signaling pathway linking the antiestrogens with MK801 is not known, the combination of the two therapy may induce the activation of both MAPK and extracellular signal-regulated kinase (ERK1/2), since the MK801 has been shown to reduce the proliferation of cancer cells through the inhibition of ERK 1/2 (104), it’s, therefore, reasonable to think that this signaling is involved in the reduction of melanoma cells growth after NMDAR antagonist treatment

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[DOI] [PubMed] [Google Scholar] 27.Goya L., Sánchez-Medina A., Redondo-Puente M., Dupak R., Bravo L., Sarriá B