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The mechanisms may lie in that the most immature and inefficient tumor blood vessels was ‘‘pruned’’ from the tumor vascular network by antiangiogenic therapy, leaving a more efficient system (192)

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We classified as high-impact variants those predicted as start-lost, stop-gained, stop-lost, splice-donor, splice-acceptor or frameshift, collectively called loss-of-function variants, whereas moderate impact variants are those predicted to otherwise affect coding or splicing of a protein (missense)