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HRD1 is involved in endoplasmic reticulum-associated degradation pathways and is responsible for labeling misfolded Aβ 42 oligomers upregulate HRD1 expression through an XBP-1 s-dependent pathway, and HRD1, as a ubiquitin ligase, decreases BACE1 expression and activity, thereby reducing Aβ 42 oligomer production and associated toxicity, and thus alleviating the associated pathology

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α-tocopherol transfer protein (TTP), which is abundant in the brain, regulates α-tocopherol levels and distribution (107)