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CDP and RDP as Alternatives to TPhP: Comparative Effects on Glucose Homeostasis and Hepatic and Whole-Pancreas Molecular Endpoints in Mice

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Prenatal and early postnatal challenges—such as abnormalities in the migration of neural crest cells, mechanical constraints during the development of the abdominal cavity, disruptions in vascular supply, or birth trauma resulting in ischemia—can lead to congenital or acquired defects in the architecture and function of the ENS [5,6,7,8]